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Diazepam is a benzodiazepine (ben-zoe-dye-AZE-eh-peens). It affects chemicals in the brain that may be unbalanced in people with anxiety. Diazepam is used to treat anxiety disorders, alcohol withdrawal symptoms, or muscle spasms. Diazepam is sometimes used with other medications to treat seizures.
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Diazepam 10mg for sleeping 2 days after 1mg/kg of cocaine.[48] The benzodiazepines are known for being able to rapidly suppress GABA neurotransmission, however they can work synergistically on some GABA agonists. 4 Neurology 4.1. GABAergic Neurotransmission One study noted an increase in GABA receptors after intravenous administration (in vivo) of diazepam (100mg/kg) in rats and guinea pigs[49] which can be mimicked by the addition of ethanol.[50] This increase may have led to an increase in neuronal excitability and could be due to increased dopamine synthesis and release, as higher concentrations of GABA were seen with ethanol[50] and other GABA agonists such as kava appear to Diazepam 5mg 30 pills US$ 150.00 US$ 5.00 have similar effects on GABA receptor mRNA[51] and neurons may be more sensitive to GABA effects than dopaminergic ones[52] and this could account for these effects. Alcohol can increase neurotransmitter secretion diazepam vs lorazepam for sleep (in the form of dopamine and glutamate) secondary to being an agonist upon metabotropic glutamate receptors.[53] May increase the sensitivity of GABA receptor signalling to agonists, with no direct interaction glutamate receptors One rat study noted that the addition of diazepam in a dose 0.1-0.5mg/kg and ethanol in doses up to 90mg/kg may not be additive due to the effects of ethanol being slightly delayed while that alcohol dose appeared additive to diazepam. 4.2. Adrenergic Neurotransmission 1mg/kg Diazepam injections (injected intragastrically) for 5 days alongside 3mg/kg Diazepam appears to increase activity of a protein known as NGF, to the same extent as 6-APB.[54] 4.3. Serotonergic Neurotransmission A suppression of 5-HT 2A and 2B receptors, the latter of which seems to be due increased 5-HT synthesis (but it is not totally reversible[55] which may explain the synergism between caffeine and 5-HT 2A antagonists) has been noted with Diazepam and ethanol in rats over two days (doses up to 20mg/kg Diazepam and pharmacy online discount 80mg/kg ethanol)[56] which can occur in vitro with ethanol as well.[57] May modify 5-HT 2A and 2B receptor signalling, but with no further research to support the theory (dietary supplementation seems to not be effective at suppressing 5-HT 2A or 2B receptor expression either[57] due to the inhibition of enzymes involved) 5 Interactions with Glucose Metabolism 5.1. Diabetes Alcohol can suppress the actions of enzyme tyrosine hydroxylase[58] which catalyses the oxidation of a small amount tyrosine to dihydro-tyrosine; the of that is oxidized has a concentration dependent effect, and the presence of alcohol (in addition to the dihydropyridyl group) reduces rate of oxidation by almost 33%, which diazepam vs xanax for sleep is an additive effect with ethanol, due to ethanol having a half-life of 3-7 hours.[59] This interaction alcohol and tyrosine hydroxylase may also occur directly with the enzyme enzyme, a protein that catalyses the oxidation of both dihydropyridine and dihydrotyrosine. The addition of alcohol to enzyme reduces the amount of tyrosine oxidized via it by 40-66%, which is known as a tyrosine-hydroxylase inhibition.[59]
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